Ulin signaling is the norm, they may contribute to a chronically altered state. In this context, these normally beneficial UPR activities could lead to insulin resistance (as a consequence of persistent IRS-1 phosphorylation and insulin receptor degradation), elevated cytokine levels (via both JNK and NF-B), fatty liver (by inhibition of VLDL secretion), and other UPR-mediated effects on metabolis
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